Research articleThe pituitary-muscle axis in mdx dystrophic mice
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Linkage between growth retardation and pituitary cell morphology in a dystrophin-deficient pig model of Duchenne muscular dystrophy
2020, Growth Hormone and IGF ResearchCitation Excerpt :Generally, increased metabolic activity due to permanent muscle necrosis and regeneration, as well as low levels of physical activity in DMD children likely contribute to a reduced growth rate [11–13]. A potential involvement of the growth hormone/insulin-like growth factor 1 (GH/IGF1) system has been discussed as well [14–17], although there is no clear evidence for morphological alterations of the adenohypophysis indicating a derangement of the hypothalamic/pituitary GH/IGF1 system in human DMD patients. However, therapeutic approaches targeting the GH/IGF1 system in DMD patients are controversial.
Pelvic organ prolapse in fibulin-5 knockout mice: Pregnancy-induced changes in elastic fiber homeostasis in mouse vagina
2007, American Journal of PathologyCitation Excerpt :In many respects, pelvic organ prolapse may be considered the end result of a multifactorial process leading to destruction of vaginal wall connective tissue. Mechanisms regulating connective tissue metabolism have received much emphasis in other degenerative conditions such as abdominal aortic aneurysms,27 inguinal hernias,15,28 rheumatoid arthritis,29 and chronic obstructive lung disease.30 However, the importance of these mechanisms in mediating pelvic organ prolapse is not well defined.
Differential effects of 3,5,3'-triiodothyronine on control and mdx myoblasts and fibroblasts: Analysis by flow cytometry
1996, Experimental Cell ResearchAnabolic steroid treatment increases myofiber damage in mdx mouse muscular dystrophy
1994, Journal of the Neurological Sciences